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Also, oxidation or alkylation in the cysteinyl moiety of GSH attenuated binding affinity, suggesting that receptor-agonist interactions may be stabilized through the presence of a reduced cysteine thiol group.Citation 5 In fact, GSH and cysteine were shown to enhance glutamatergic-induced (NMDA and non-NMDA agonists) release of [ 3 H]dopamine in mouse striatal slices